Citaat:
Oorspronkelijk geplaatst door Jan van den Berghe
Ik wacht met veel ongeduld op het eerste wetenschappelijke bewijs over uw theorietje over "omgekeerde evolutie".
Waarom komt dat er maar niet?
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Ik heb net iets gevonden, dit onderzoek bevestigt mijn stelling dat lockdowns het virus dodelijker maken.
Het stelt dat menselijk gedrag een grote invloed heeft op de evolutie van COVID. Zij keken naar de virale lading en besmettelijkheid. De lockdowns duwde het virus evolutionair om meer besmettelijk worden en kreeg het een hogere virale lading waardoor het dodelijker werd. Hogere virale lading = dodelijker.
Bij deze dus, dozo, …
https://www.news-medical.net/news/20...s-strains.aspx
Many environmental factors influence this evolution, including human behavior. By isolating sick (infected) people and using lockdowns to control outbreaks, humans may alter virus evolution in complicated ways.
An important concept in this interaction is viral load, which refers to the amount or concentration of a virus present per ml of a bodily fluid. In SARS-CoV-2, a higher viral load in respiratory secretions increases the risk of transmission through droplets. Viral load relates to the potential to transmit a virus to others. For example, a virus like Ebola has an exceptionally high viral load, whereas the common cold has a low one. However, viruses must perform a careful balancing act, as increasing the maximum viral load can be advantageous, but an excessive viral load may cause individuals to become too sick to transmit the virus to others.
The results showed a clear difference. As the virus evolved from the Wuhan strain to the Delta strain, they found a 5-fold increase in the maximum viral load and a 1.5-fold increase in the number of days before the viral load peaked.
Iwami and his colleagues suggest that
human behavioral changes in response to the virus, designed to limit transmission, were increasing the selection pressure on the virus. This caused SARS-CoV-2 to be transmitted mainly during the asymptomatic and presymptomatic periods, which occur earlier in its infectious cycle. As a result, the viral load peak advanced to this period to spread more effectively in the earlier pre-symptomatic stages.
Their study suggests the possibility that new strains of coronavirus evolved because of a complex interaction between clinical symptoms and human behavior.
https://neurosciencenews.com/human-b...r-covid-25249/
Key Facts:
Human responses to COVID-19, such as isolation and lockdowns, have impacted the evolution of the virus, making it more transmissible early in infection.
The study showed a 5-fold increase in maximum viral load and a faster peak as SARS-CoV-2 evolved from the Wuhan to Delta variants.
This research highlights the need to incorporate human behavior into understanding virus evolution and in developing adaptive public health strategies.
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