[morte-vivante]

daar zitten een paar pareltjes tussen.

Uit het toxicologierapport van cannabis en resins:

Cannabis is not associated with acute fatal overdoses.
Lethality studies in animals show the doses needed to induce mortality are well beyond what could possibly be consumed by a human

There is a wealth of data from studies on cells and animals supporting the notion that cannabinoids have immunosuppressant and anti-inflammatory effects

A wealth of preclinical literature demonstrates that cannabinoids reduce cancer cell proliferation, inducing apoptosis in these cells, as well as inhibiting cancer cell migration and angiogenesis in numerous cancer cell types (19). There is moderately strong epidemiological evidence that cannabis use does not increase the risk of cancers of the lung, head and neck

No relationship could be found between the age of onset of cannabis use and cognitive function. Furthermore, no association between cannabis use and reduced cognitive function could be found in studies with a greater than 72-hour abstinence period, suggesting that the effects of cannabis use on cognition were reversible.

The argument that cannabis causes schizophrenia is contentious,
however, as some have observed that sharp increases in global cannabis use in recent decades has not increased the incidence of schizophrenia (39).
However, other studies have linked increased prevalence of cannabis use in specific localities with increased incidence of schizophrenia (40, 41). Importantly, most of the evidence that cannabis causes schizophrenia comes from studies of during-adolescence users, and adolescence is the period of highest risk for developing schizophrenia. The rates of cannabis-induced psychosis may be lower in patients who commence cannabis use in adulthood. The vast majority of people who use cannabis will never develop a psychotic disorder, and those who do are likely to have some genetic vulnerability to cannabis-induced psychosis

1.9 Driving under the influence of drugs

There is an array of evidence to support the idea that people driving under the influence of cannabis are more likely to be involved in a car accident (reviewed in (1)), although the level of risk is generally not as great as with alcohol (31). A large systematic review that incorporated results for 239739 participants from 21 case–control and culpability studies in 13 countries, showed that cannabis use caused a low-to-moderate (10-30%) increase of being in an accident (1, 44).The relatively low risk may be due to cannabis users overestimating their level of impairment and recruiting strategies to compensate for the effects of cannabis on their driving performance (45). By contrast, alcohol-intoxicated individuals underestimate their level of impairment. Laboratory studies show that cannabis acutely impairs certain types of cognitive function and psychomotor skills and can diminish driving performance under certain conditions (46, 47). These cognitive and performance deficits are less apparent in experienced cannabis users due to tolerance, and may even be absent (48). Some studies suggest that drivers under the influence of cannabis drive more slowly, make fewer attempts to overtake, and leave greater distances between themselves and the vehicle in front (49). However, other studies have shown that cannabis use impairs reaction time, lane control, speedometer monitoring, hand and body steadiness and braking time as well as promoting inappropriate responses in an emergency scenario (50–53)

Neveneffecten voor cannabis:

Cannabis consumption causes euphoria, laughter and talkativeness. It is an appetite stimulant, and may promote dry mouth and dizziness as well as increasing visual, olfactory and auditory perceptions